Prostate Cancer

Carotenoids

1.Dietary Carotenoid Intakes and Prostate Cancer Risk: A Case-Control Study from Vietnam

2.Effect of Carotene and Lycopene on the Risk of Prostate Cancer: A Systematic Review and Dose-Response Meta-Analysis of Observational Studies

3.Lycopene and Risk of Prostate Cancer: A Systematic Review and Meta-Analysis

4.Antitumor Effects of Saffron-Derived Carotenoids in Prostate Cancer Cell Models

5.A Meta-Analysis of Dietary Carotenoids and Prostate Cancer Incidence

6.Serum Lycopene, Other Carotenoids, and Prostate Cancer Risk: a Nested Case-Control Study in the Prostate, Lung, Colorectal, and Ovarian Cancer Screening Trial

Epigallocatechingallate (EGCG)

1.Green tea polyphenols for prostate cancer chemoprevention: A translational perspective

2.Molecular Targets for Green Tea in Prostate Cancer Prevention

Honokiol

1.C-Mycis a novel target of cell cycle arrest by honokiol in prostate cancer cells

2.Honokiol Activates Reactive Oxygen Species-Mediated Cytoprotective Autophagy in Human Prostate Cancer Cells

3.Honokiol, a constituent of Magnolia species, inhibits adrenergic contraction of human prostate strips and induces stromal cell death

4.Honokiol Inhibits Androgen Receptor Activity in Prostate Cancer Cells

5.Honokiol causes G0-G1 phase cell cycle arrest in human prostate cancer cells in association with suppression of retinoblastoma protein level/ phosphorylation and inhibition of E2F1 transcriptional activity

6.Honokiol, a Natural Plant Product, Inhibits the Bone Metastatic Growth of Human Prostate Cancer Cells

Isoflavones

1.Isoflavones and Prostate Cancer: A Review of Some Critical Issues

2.Phase II trial of isoflavone in prostate-specific antigen recurrent prostate cancer after previous local therapy

Luteolin

1.Inhibition of ANO1 by luteolin and its cytotoxicity in human prostate cancer PC-3 cells

2.Luteolin inhibited proliferation and induced apoptosis of prostate cancer cells through miR-301

3.Gefitinib and Luteolin Cause Growth Arrest of Human Prostate Cancer PC-3 Cells via Inhibition of Cyclin G Associated Kinase and Induction of miR-630

4.Luteolin Inhibits Human Prostate Tumor Growth by Suppressing Vascular Endothelial Growth Factor Receptor 2-Mediated Angiogenesis

5.Luteolin inhibits invasion of prostate cancer PC3 cells through E-cadherin

Lycopene

In Vitro

1.Lycopene Treatment of Prostate Cancer Cell Lines Inhibits Adhesion and Migration Properties of the Cells

2.Effects of Lycopene on Protein Expression in Human Primary Prostatic Epithelial Cells

3.Lycopene induces cell growth inhibition by altering mevalonate pathway and Ras signaling in cancer cell lines

In Vivo

1.Tomatoes or Lycopene: a Role in Prostate Carcinogenesis?

Clinical Trials

1. Tomato consumption and prostate cancer risk: a systematic review and meta-analysis

2. Tomato-based randomized controlled trial in prostate cancer patients: Effect on PSA

3. Lycopene and Risk of Prostate Cancer

4. Effect of Carotene and Lycopene on the Risk of Prostate Cancer: A Systematic Review and Dose-Response Meta-Analysis of Observational Studies

5. The Potential Role of Lycopene for the Prevention and Therapy of Prostate Cancer: From Molecular Mechanisms to Clinical Evidence

6. Lycopene, Tomato Products, and Prostate Cancer Incidence: A Review and Reassessment in the PSA Screening Era

7. Lycopene/Tomato Consumption and the Risk of Prostate Cancer: A Systematic Review and Meta-analysis of Prospective Studies

8. Tomato Products, Lycopene, and Prostate Cancer: A Review of the Epidemiological Literature

9. Lycopene and prostate cancer

10. Tomato Phytochemicals and Prostate Cancer Risk

11. Tomatoes, Lycopene, and Prostate Cancer: Progress and Promise

12. Can the Consumption of Tomatoes or Lycopene Reduce Cancer Risk?

13. A Prospective Study of Tomato Products, Lycopene, and Prostate Cancer Risk

14. Lycopene and prostate cancer

15. Tomatoes, Tomato-Based Products, Lycopene, and Cancer: Review of the Epidemiologic Literature

16. Effects of Lycopene Supplementation in Patients with Localized Prostate Cancer

Panax ginseng

1.Anticancer activity and potential mechanisms of 1C, a ginseng saponin derivative, on prostate cancer cells

2.Increase in apoptotic effect of Panax ginseng by microwave processing in human prostate cancer cells: in vitro and in vivo studies

3.Anti-proliferative effect of ginseng saponins on human prostate cancer cell line

Resveratrol

In Vitro

1.Resveratrol- activates autophagic cell death in prostate cancer cells via down regulation of STIM1 and the mTOR pathway

2.Resveratrol induces cell cycle arrest and apoptosis with docetaxel in prostate cancer cells via a p53/ p21WAF1/CIP1 and p27KIP1 pathway

3.Resveratrol directly targets DDX5 resulting in suppression of the mTORC1 pathway in prostate cancer

4.Resveratrol suppresses the epithelial-to-mesenchymal transition in PC-3 cells by down-regulating the PI3K/AKT signaling pathway

5. Resveratrol Activates Autophagic Cell Death in Prostate Cancer Cells via Downregulation of STIM1 and the mTOR Pathway

6. Resveratrol Inhibits IL-6-Induced Transcriptional Activity of AR and STAT3 in Human Prostate Cancer LNCaP-FGC Cells

7. A novel anti cancer effect of resveratrol: reversal of epithelial mesenchymal transition in prostate cancer cells

8. Resveratrol enhances radiation sensitivity in prostate cancer by inhibiting cell proliferation and promoting cell senescence and apoptosis

9. Resveratrol enhances prostate cancer cell response to ionizing radiation. Modulation of theAMPK, Akt and mTOR pathways

10. Combination of Resveratrol and Antiandrogen Flutamide Has Synergistic Effect on Androgen Receptor Inhibition in Prostate Cancer Cells

11. Resveratrol Induces Growth Arrest and Apoptosis through Activation of FOXO Transcription Factors in Prostate Cancer Cells

12. Antiproliferative Effects of Resveratrol and the Mediating Role of Resveratrol Targeting Protein NQO2 in Androgen Receptor positive, Hormone-non-responsive CWR22Rv1 Cells

13. Repressive Effects of Resveratrol on Androgen Receptor Transcriptional Activity

14. Resveratrol Down-Regulates the Androgen Receptor at the Post-Translational Level in Prostate Cancer Cells

15. Mechanisms Involved in Resveratrol-Induced Apoptosis and Cell Cycle Arrest in Prostate Cancer–Derived Cell Lines

16. Resveratrol-caused apoptosis of human prostate carcinoma LNCaP cells is mediated via modulation of phosphatidylinositol 3’-kinase/Akt pathway and Bcl-2 family proteins

17. Resveratrol Induces Prostate Cancer Cell Entry into S Phase and Inhibits DNA Synthesis

18. Resveratrol Inhibits the Expression and Function of the Androgen Receptor in LNCaP Prostate Cancer Cells

In Vivo

1. Resveratrol and pterostilbene epigenetically restore PTEN expression by targeting oncomiRs of the miR-17 family in prostate cancer

2. Resveratrol regulates PTEN/Akt pathway through inhibition of MTA1/HDAC unit of the NuRD complex in prostate cancer

3. Resveratrol–zinc combination for prostate cancer management

4. Dietary Resveratrol prevents development of high-grade prostatic intraepithelial neoplastic lesions: Involvement of SIRT1/S6K axis

5. Resveratrol Reduces Prostate Cancer Growth and Metastasis by Inhibiting the Akt/MicroRNA-21 Pathway

6. Apoptosis by dietary agents for prevention and treatment of prostate cancer

7. Suppression of Prostate Cancer Growth by Resveratrol in The Transgenic Rat for Adenocarcinoma of Prostate (TRAP) Model

8. Resveratrol suppresses prostate cancer progression in transgenic mice

Silymarin

1. Molecular Mechanisms of Silibinin-Mediated Cancer Chemoprevention with Major Emphasis on Prostate Cancer

2. Angiopreventive Efficacy of Pure Flavonolignans from Milk Thistle Extract against Prostate Cancer: Targeting VEGF-VEGFR Signaling

Vitamin C

1. Weekly ascorbic acid infusion in castration-resistant prostate cancer patients: a single-arm phase II trial

2. Intravenous Vitamin C and Metabolic Correction as Adjuvant Therapy for prostate Cancer: a Case Report

3. Association between Dietary Vitamin C Intake and Risk of Prostate Cancer: A Meta-analysis Involving 103,658Subjects

4. Elimination of Ascorbic Acid After High-Dose Infusion in Prostate Cancer Patients: A Pharmacokinetic Evaluation